Memory T cell - Wikipedia. A lymphocyte is shown in the center of this picture. After the naive T cell (N) encounters an antigen it becomes activated and begins to proliferate (divide) into many clones or daughter cells. Some of the T cell clones will differentiate into effector T cells (E) that will perform the function of that cell (e. Some of the cells will form memory T cells (M) that will survive in an inactive state in the host for a long period of time until they re- encounter the same antigen and reactivate.

Memory T cells are a subset of infection- as well as potentially cancer- fighting T cells (also known as a T lymphocyte) that have previously encountered and responded to their cognate antigen; thus, the term antigen- experienced T cell is often applied. Such T cells can recognize foreign invaders, such as bacteria or viruses, as well as cancer cells. Memory T cells have become . At a second encounter with the invader, memory T cells can reproduce to mount a faster and stronger immune response than the first time the immune system responded to the invader. This behaviour is utilized in T lymphocyte proliferation assays, which can reveal exposure to specific antigens. Sub- populations. The single unifying theme for all memory T cell subtypes is that they are long- lived and can quickly expand to large numbers of effector T cells upon re- exposure to their cognate antigen.

By this mechanism they provide the immune system with . Memory T cells may be either CD4+ or CD8+ and usually express CD4. RO. Central memory T cells also have intermediate to high expression of CD4. This memory subpopulation is commonly found in the lymph nodes and in the peripheral circulation. They also have intermediate to high expression of CD4. These memory T cells lack lymph node- homing receptors and are thus found in the peripheral circulation and tissues.

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One cell surface marker that has been associated with TRM is the integrin . These cells are thought to play a major role in protective immunity against pathogens. Thus, although this population as a whole is abundant within the peripheral circulation, individual virtual memory T cell clones reside at relatively low frequencies. One theory is that homeostatic proliferation gives rise to this T cell population. Although CD8 virtual memory T cells were the first to be described. For example, in the mouse, Sendai virus specific CD8+ T- cells low on CD4.

CD8 T- cells can also be distinguished from activated effector CD8 T- cells using CD4. Other investigators have studied Stem memory TSCM cells.

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• Memory T cells are a subset of infection- as well as potentially cancer-fighting T cells (also known as a T lymphocyte) that have previously encountered.
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Like naive T cells, TSCM cells are CD4. RO. Although most information is currently based on observations in the cytotoxic T cells (CD8- positive) subset, similar populations appear to exist for both the helper T cells (CD4- positive) and the cytotoxic T cells.

TCM). The TCM cells are thought to contain some properties associated with memory cells stem cells. TCM display a capacity for self- renewal due to high levels of phosphorylation of an important transcription factor known as STAT5. In preclinical studies, adoptively transferred TSCM confer superior immunity compared with other T memory subsets. Journal of Immunology. Immunity & Ageing. Immunological Reviews. Journal of Autoimmunity.

Nature Reviews Immunology. Activation phenotype, rather than central– or effector–memory phenotype, predicts the recall efficacy of memory CD8+ T cells.

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Proceedings of the National Academy of Sciences of the United States of America. Immunobiology: the immune system in health and disease. New York: Garland Science. ISBN 9. 78- 0- 4. Lichtman, Andrew H.; Abbas, Abul K.

Cellular and molecular immunology. Philadelphia: Saunders.

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